Mito Group

Mito Group company information, Employees & Contact Information

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We work in four Business Units: Mito Creative on branding and campaigns from strategy to execution, Mito Digital on human-centered interfaces for enterprises of the digital era, Mito Performance on PPC, media, programmatic, SEO, CRO, analytics and research, while Mito Next on service design, data-driven marketing and marketing technologies.

Company Details

Employees
293
Address
Károlyi Str. 9., Budapest,magyarország 1053,hungary
Phone
+36 30 248 7938
Email
in****@****mito.hu
Industry
Professional Services
NAICS
Professional, Scientific, and Technical Services
Architectural, Engineering, and Related Services
Other Professional, Scientific, and Technical Services
Landscape Architectural Services
Engineering Services
Drafting Services
Building Inspection Services
Geophysical Surveying and Mapping Services
Surveying and Mapping (except Geophysical) Services
Testing Laboratories
All Other Professional, Scientific, and Technical Services
Website
mito.hu
HQ
Budapest, Magyarország
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Mito Group Questions

News

Biomimetic Hydrogen-Generating Piezoelectric Hydrogel for Dual Glucose Regulation and Mitophagy Activation in Diabetes-Induced IVDD - Wiley

Biomimetic Hydrogen-Generating Piezoelectric Hydrogel for Dual Glucose Regulation and Mitophagy Activation in Diabetes-Induced IVDD Wiley

Peripheral mitochondrial transplantation alleviates diabetes-associated cognitive dysfunction by suppressing cuproptosis - ScienceDirect.com

Peripheral mitochondrial transplantation alleviates diabetes-associated cognitive dysfunction by suppressing cuproptosis ScienceDirect.com

Boosting neuronal activity-driven mitochondrial DNA transcription improves cognition in aged mice - Science | AAAS

Boosting neuronal activity-driven mitochondrial DNA transcription improves cognition in aged mice Science | AAAS

Mitochondria-targeted hydroxyurea inhibits OXPHOS and induces antiproliferative and immunomodulatory effects - ScienceDirect.com

Mitochondria-targeted hydroxyurea inhibits OXPHOS and induces antiproliferative and immunomodulatory effects ScienceDirect.com

Mitophagy in ototoxicity - Frontiers

Mitophagy in ototoxicity Frontiers

Stabilization of HIF-1α alleviates osteoarthritis via enhancing mitophagy - Nature

Stabilization of HIF-1α alleviates osteoarthritis via enhancing mitophagy Nature

Survivors’ Stories: Tomie Mito, 106, Fuchu-cho, Hiroshima―Her father died in the atomic bombing, and her unborn child was exposed - 中国新聞ヒロシマ平和メディアセンター

Survivors’ Stories: Tomie Mito, 106, Fuchu-cho, Hiroshima―Her father died in the atomic bombing, and her unborn child was exposed 中国新聞ヒロシマ平和メディアセンター

Mitophagy defect mediates the aging-associated hallmarks in Hutchinson–Gilford progeria syndrome - Wiley Online Library

Mitophagy defect mediates the aging-associated hallmarks in Hutchinson–Gilford progeria syndrome Wiley Online Library

Mitophagy associated self-degradation of phosphorylated MAP4 guarantees the migration and proliferation responses of keratinocytes to hypoxia | Cell Death Discovery - Nature

Mitophagy associated self-degradation of phosphorylated MAP4 guarantees the migration and proliferation responses of keratinocytes to hypoxia | Cell Death Discovery Nature

Mitocytosis, a migrasome-mediated mitochondrial quality-control process - ScienceDirect.com

Mitocytosis, a migrasome-mediated mitochondrial quality-control process ScienceDirect.com

Mic60/Mitofilin determines MICOS assembly essential for mitochondrial dynamics and mtDNA nucleoid organization - Nature

Mic60/Mitofilin determines MICOS assembly essential for mitochondrial dynamics and mtDNA nucleoid organization Nature

Mitochondria-Targeted Cancer Therapy Using a Light-Up Probe with Aggregation-Induced-Emission Characteristics - Wiley Online Library

Mitochondria-Targeted Cancer Therapy Using a Light-Up Probe with Aggregation-Induced-Emission Characteristics Wiley Online Library

Ripk3 induces mitochondrial apoptosis via inhibition of FUNDC1 mitophagy in cardiac IR injury - ScienceDirect.com

Ripk3 induces mitochondrial apoptosis via inhibition of FUNDC1 mitophagy in cardiac IR injury ScienceDirect.com

VCP recruitment to mitochondria causes mitophagy impairment and neurodegeneration in models of Huntington’s disease - Nature

VCP recruitment to mitochondria causes mitophagy impairment and neurodegeneration in models of Huntington’s disease Nature

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