Assistant in classes, lead discussion sessions and test correction Disciplines: Cell Biology (3 quarters), Introduction to Human Heredity (2 quarters), Genes and Genetics (1 quarter)

Dr. Fawaz Haj’s Lab – Nutrition Department - UCDavis - Feb 2015 – Dez 2019Studies of the molecular basis of metabolic diseases with a focus on the liver contribution to the systemic homeostasis and physiology. Using small animal models, biochemistry, cellular and molecular biology techniques.Project 1: Soluble epoxide hydrolase is a potential therapeutic target for non-alcoholic steatohepatitisProject 2: Soluble epoxide hydrolase is a potential therapeutic target for… Show more Dr. Fawaz Haj’s Lab – Nutrition Department - UCDavis - Feb 2015 – Dez 2019Studies of the molecular basis of metabolic diseases with a focus on the liver contribution to the systemic homeostasis and physiology. Using small animal models, biochemistry, cellular and molecular biology techniques.Project 1: Soluble epoxide hydrolase is a potential therapeutic target for non-alcoholic steatohepatitisProject 2: Soluble epoxide hydrolase is a potential therapeutic target for alcoholic steatohepatitisProject 3: Soluble epoxide hydrolase role in acute liver injury Show less

Trainee of NIH Superfund Research Program at UCDavis Project 5 : Endoplasmic Reticulum Stress Within the goals of the UC Davis Superfund Center, this project investigate new mechanistic insights into the effects of chronic exposure of Superfund (SF) chemicals on endoplasmic reticulum (ER) stress, which leads to fibrosis and organ damage. Recent and exciting data demonstrate that exposure to xenobiotics perturbs the levels of P450-generated lipid mediators, which directly affect the pathological… Show more Trainee of NIH Superfund Research Program at UCDavis Project 5 : Endoplasmic Reticulum Stress Within the goals of the UC Davis Superfund Center, this project investigate new mechanistic insights into the effects of chronic exposure of Superfund (SF) chemicals on endoplasmic reticulum (ER) stress, which leads to fibrosis and organ damage. Recent and exciting data demonstrate that exposure to xenobiotics perturbs the levels of P450-generated lipid mediators, which directly affect the pathological development of ER stress. Thus, this project directly test the novel hypothesis that plasma lipid mediators may represent a surrogate biomarker for ER stress following chronic xenobiotic exposure, as well as developing a new cell-based bioassay for quantitation of pro-ER stress environmental toxicants in field samples. Show less