Worked on two projects:1. Apo-dystrophin Dp116, Dp71, Dp40 and Dp30: localisation and expression during early embryo-genesis Abstract. Dystrophin and its apo-forms are structural proteins that link intracellular actin scaffolding of the cell with intercellular space. They have been recognized as the main factor in protecting and holding the sarcolemma intact after it has been subjected to great stress. Actin is connected to dystrophin, which forms a multistage complex called dystrophin-associated protein complex (DAPC) with laminin 2 in the extracellular space. Muscular dystrophy is a hereditary disorder caused by progressive skeletal weakness based on defects in the sarcolemma, leading to cell death and inability to recover after simple physical activity. Despite greater understanding of the causes of muscular dystrophy, the role of DAPC in the development of the disease is still unclear. Using immunohistochemistry and western blotting, this study attempts to shed light on the expression of apo-dystrophin in different stages of embryogenesis. The results find programmed expressions of different types of apo-dystrophin at different stages of embryonic development of skeletal and cardiac muscle in a variety of mutants. 2. Post-transcriptional modifications of mRNA. NSUN6 and its biological role in the cell cycle Abstract for the second paper is uploaded on this page

I was performing the surgical procedure of putting rats in hemorrhagic shock

Resistant TB in South African population

Mobile clinic that provided necessary medicine for preventable diseases in the poorest area of Colombo and the rest of the country

Treatment of patients with acute cardiac and brain diseases also include trauma patients