I am working as a manager/operator in the flow cytometry and high-speed cell sorting facility at Auburn University College of Veterinary medicine.

After moved on to Associate professor, I started teaching immunology lecture and veterinary hygiene lab course for whole semester. In addition to teaching in the class, I started mentoring senior students in the lab who need to complete their graduation thesis in 3 years. I worked with 10 students since I moved to Yamaguchi and could publish several reports to peer reviewed scientific journal.

After experiencing postdoc in the US, I moved to Japan to start a career as a faculty in the national university. I was assigned as an assistant professor in the department of veterinary medicine at Tokyo University of Agriculture and Technology. In Japan, veterinary medicine is taught in 6 years course and every students have finish two or three years lab projects to write graduation thesis. I supervised two students and wrapped up obtained results in a scientific report.

To gain deep insight into molecular virology in combination with cellular biology, I moved to the US and started to work as a postdoctoral fellow in Dr. Robert Lamb's lab at Northwestern University, Howard Hughes Medical Institute (HHMI). I was involved in a project focusing on the cellular mechanism that supports the influenza virus life cycle at Northwestern University.

After 1 year training as a research resident supported by Japan Health and Science Foundation, I had started new position at NIID, as a research scientist who is in charge of the detection, identification, education, and basic research related to severe acute respiratory syndrome (SARS) coronavirus (SARS-CoV). I got a training as a specialist working with BSL3 pathogen at P3 lab. In this period, I had examined the possibility for the emergence of highly-virulent SARS-CoV by using basic molecular virology approach. This project was published on Journal of Virology in 2008, titled as "Entry from the cell surface of SARS-CoV with cleaved S protein".In addition, I had collaborated with senior research scientist to develop the animal model for SARS-CoV infection. This project was published on Microbiology and Immunology in 2008, titled as "Co-infection of respiratory bacterium with severe acute respiratory syndrome coronavirus induces an exacerbated pneumonia in mice.n".

As NIID accepted the senior research student from nearby college, I had supervised them each year to complete their senior research project.

After getting PhD, I had started the training as a research resident supported by Japan Health and Science Foundation (JHSF). JHSF sent me to the lab at National Institute of Infectious Diseases, Japan. I was working on mouse hepatitis coronavirus to clarify the mechanism of "receptor-independent infection". "Receptor-independent infection" is one of the common feature of the viruses that display high virulence/mortality. I could show the correlation between one amino acid mutation, fragility of the spike protein that is an essential protein to start virus infection, and the ability to complete "receptor-independent infection". This project was published on Journal of Virology, titled as "Receptor-independent infection of murine coronavirus: analysis by spinoculation" in 2006. This project was further proceeded and another report was published on Virology, titled as "Heparan sulfate is a binding molecule but not a receptor for CEACAM1-independent infection of murine coronavirus" in 2007.