-Led in-house studies of intrahippocampal kainic acid epilepsy model in rodents to evaluate in vivo efficacy of CODA's chemogenetic therapeutics-Worked to implement in-house EEG recording equipment to be able to record seizures in awake, behaving mice.-Conducted stereotaxic delivery of CODA's AAV gene therapy and EEG electrode placement to assess efficacy of CODA's gene therapy platform on seizure burden.-Led efforts on implementing EEG data collection, analysis, and interpretation of results to help guide CODA's gene therapy development for use in clinical settings.-Managed Research Associates to assist with surgeries, data collection, and data analysis.

-Led project investigating how seizure induced activation of the hypothalamic-pituitary-adrenal (HPA) axis worsens neuropathology, contributes to co-morbid mood disorders, and worsens seizure burden in a mouse model of epilepsy. -Performed stereotaxic surgery to deliver intrahippocampal kainic acid and chemogenetic vectors.-Implanted EEG headmounts with local field potential electrode to record EEG in awake, behaving mice to detect seizures.-Conducted a battery of behavioral assays to test for anxiety- and depression-like states in epileptic mice.-Used immunofluorescence assays to assess neuropathological damage and neurodegeneration following chronic epilepsy.-Ran corticosterone ELISAs to detect changes in circulating corticosterone during epileptogenesis.-Independently collected, analyzed, interpreted, and presented data produced from the project.

PhD Candidate in the Neuroscience Training ProgramMy thesis work in Dr. Avtar Roopra's laboratory focused on understanding the role for class I Histone Deacetylases (HDACs) in contributing to mTOR dependent synaptic plasticity deficits and a novel decreased seizure threshold phenotype in a mouse model of Tuberous Sclerosis Complex (TSC).-Performed electrophysiology to record long term depression (LTD) and long term potentiation (LTP) in hippocampal slices bath treated with histone deacetylase inhibitors-Modeled epilepsy in rodents using 1) intraperitoneal delivery of kainic acid 2) flurothyl -Used an array of molecular biology techniques (western blotting, qRTPCR, immunofluorescence, protein immunoprecipitation, and chromatin immunoprecipitation) to asses impacts of histone modifiers on synaptic plasticity and seizure burden-Conducted behavioral assays in genetic mouse model of epilepsy to determine therapeutic efficacy of histone modifying drugs on aberrant behavior and seizure threshold.-Independently collected, analyzed, and interpreted data.-Extensive experience presenting data at conferences and invited speaker positions.

As an undergraduate research assistant in Dr. Michael Ragozzino's laboratory, I helped generate data in understanding the differential role of 5-HT2A and 5-HT2C receptors in behavioral inflexibility and working memory.