Jordan Sheets, Phd, Ms Email & Phone Number

California, United States

As the Chief Executive Officer of Origin Learning Fund (OLF), I am dedicated to driving positive change through educational empowerment. OLF, a 501(c)3 nonprofit organization, is committed to making education accessible to underserved communities by leveraging advanced technologies and resources.In this volunteer role, I provide visionary leadership, strategic direction, and operational oversight, ensuring that OLF fulfills its mission to bridge educational gaps and uplift communities hindered by socioeconomic disparities. I work closely with our dedicated team to design and implement initiatives that create real-world impact, fostering opportunities for growth and development.

Hercules, Ca, Us

Hercules, Ca, Us

• Serving as chief expert in applications support surrounding the QX One instrument (Bio-Rad’s ‘flagship instrument’) • Develop tools to coordinate effective and consistent training experiences between the FASs and customers • Collaborate and coordinate with Bio-Rad’s Marketing Team, Digital Biology Group, Technical Support Team, and Software/Engineering Team to resolve important instrument ‘bugs’ and ‘errors’ • Organize and communicate important updates to the FAS organization concerning the developmental progress of software/firmware/instrumentation • Resolve escalated customer cases surrounding QX One performance in key accounts

Hercules, Ca, Us

• Consulting customers in pre and post-sales support to provide objective and technical expertise, propelling scientific research projects forward toward success • Utilizing pre-existing laboratory technical skills and education to perform ‘bench work’, troubleshooting, and in-depth analysis of various genomic-based methodologies including Next Generation Sequencing (NGS), droplet digital PCR (ddPCR), and real-time quantitative PCR (RT-qPCR) • Lead trainings, demonstrations, and seminars for supported customer products (on-site or virtual) • Train and mentor new members of the FAS team as well as sales colleagues • Work with customers to analyze anomalous data; troubleshooting to improve workflow • Provide feedback to the sales and marketing group on current and emerging market opportunities for Cell and Gene Therapy sectors

Hercules, Ca, Us

Sioux Falls, Us

Brief Synopsis of Project: CLN6 disease belongs to a family of rare, fatal neurodegenerative disorders collectively called neuronal ceroid lipofuscinoses (NCLs) for which no effective treatment currently exists. While affected cell types display accumulation of lysosomal storage material and neuronal degeneration, the mechanisms underlying this pathology is poorly understood. Animal models of CLN6 disease and human autopsies have revealed a correlation between regions of neuronal loss and activation of astrocytes and microglia, suggesting that gliosis may play a role in neuronal degeneration. The current project seeks to identify the role of gliosis in the progression of CLN6 disease. Cortical neurons and glia were harvested from Cln6-deficient and wild type (Cln6nclf and WT, respectively) mice at postnatal day 1 (P1) and postnatal day 2 (P2) for in vitro analyses. Utilizing ICC, I quantified the relative intensity of NCL hallmark markers (LysoTracker, ATP synthase subunit C, and autofluorescent storage material) in primary neuronal cultures. I have reported significant differences in NCL markers between Cln6nclf (designated Cln6 henceforth) and WT neuronal cells after maintaining cultures for 14 days in vitro (DIV14); decreased staining intensity of LysoTracker red and increased accumulation of ATP synthase subunit and ASM in Cln6 neurons was observed in comparison with staining intensities of WT neurons. To define the effect of Cln6- deficiency on the activation status of glia, I have isolated populations of resting astrocytes and microglia and immunostained them with activation markers glial fibrillary acidic protein (GFAP) and CD68, respectively. Resting Cln6 astrocytes and microglia displayed significantly higher intensities of glial fibrillary acidic protein (GFAP) and CD68 immunostaining, respectively, compared to resting WT glia, suggesting that Cln6-deficiency in glia predicts pre-mature activation.

Spearfish, Sd, Us

Teaching Assistantship – Worked underneath the supervision of Dr. Garth Spellman (assistant professor); collaborated on curriculum and exam development, met with students upon request, and graded all written work, including final exam papers

Doctor Of Philosophy - Phd, Basic Biomedical Sciences (Cancer Biology Emphasis)

Mini-Mba, Marketing

Master'S Degree, Integrative Genomics